Brandon Schneider
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70f9b10127
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ingest: MS myelin glucose signaling article (2026-05-04)
Brain glucose levels regulate OPC fate: high glucose → proliferation,
low glucose → maturation. Acetyl-CoA from glucose drives histone
acetylation for OPC division; ketone bodies substitute for myelin
synthesis. Ketogenic diet rescues myelin in ACLY-deficient mice.
Connects to: N-Dimensional Gene Hypothesis, PIST polymorphic shifter,
topological state machine, FAMM delay lines, waveprobe manifolds
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2026-05-06 23:53:20 -05:00 |
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