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Brain glucose levels regulate OPC fate: high glucose → proliferation, low glucose → maturation. Acetyl-CoA from glucose drives histone acetylation for OPC division; ketone bodies substitute for myelin synthesis. Ketogenic diet rescues myelin in ACLY-deficient mice. Connects to: N-Dimensional Gene Hypothesis, PIST polymorphic shifter, topological state machine, FAMM delay lines, waveprobe manifolds
30 lines
1 KiB
Text
30 lines
1 KiB
Text
created: 20260506230000000
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modified: 20260506230000000
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tags: ResearchStack Compression Hutter CAD Geometry PIST Shells
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title: Pathological PIST Shells
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type: text/vnd.tiddlywiki
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! Pathological PIST Shells
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Concentric polygonal rings representing PIST shell encoding. Each shell k is
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drawn as a regular (2k+1)-gon with radius proportional to k. Mirror involution
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axes are marked at the t = k symmetry points (red markers).
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Inner shells are dense with low curvature; outer shells are sparse with high
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curvature — this is the tension gradient used in compression. The mirror
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involution (k, t) → (k, 2k+1-t) preserves mass and is self-inverse, providing
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the geometric basis for resonance jumps and shell-preserving RG flow.
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One of three pathological manifold structures (Torus-Menger-Horn) for Hutter
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Prize compression folding.
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!! Links
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* [[PIST Shifters]]
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* [[Hutter Prize Compression]]
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* [[Pathological Manifold Torus]]
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* [[Pathological Menger-Horn Composite]]
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!! Durable Source
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`../../3-Mathematical-Models/cad_models/pist_shells.scad`
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