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Brain glucose levels regulate OPC fate: high glucose → proliferation, low glucose → maturation. Acetyl-CoA from glucose drives histone acetylation for OPC division; ketone bodies substitute for myelin synthesis. Ketogenic diet rescues myelin in ACLY-deficient mice. Connects to: N-Dimensional Gene Hypothesis, PIST polymorphic shifter, topological state machine, FAMM delay lines, waveprobe manifolds
24 lines
781 B
Text
24 lines
781 B
Text
created: 20260506230000000
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modified: 20260506230000000
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tags: ResearchStack Compression Hutter CAD Geometry Torus
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title: Pathological Manifold Torus
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type: text/vnd.tiddlywiki
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! Pathological Manifold Torus
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3D slice of a hypertorus used as a PIST pathological manifold for Hutter Prize
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compression geometry. A 3D projection of the 4D structure, with points sampled
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via irrational φ-rotations (Φ, Φ², Φ³) and the psi angle encoded as color.
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The torus provides periodic boundary conditions for routing across the
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compression manifold — no endpoints, no zero-mass singularities.
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!! Links
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* [[Hutter Prize Compression]]
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* [[Pathological Menger-Horn Composite]]
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* [[Pathological PIST Shells]]
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!! Durable Source
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`../../3-Mathematical-Models/cad_models/hypertorus_slice.scad`
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