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Brain glucose levels regulate OPC fate: high glucose → proliferation, low glucose → maturation. Acetyl-CoA from glucose drives histone acetylation for OPC division; ketone bodies substitute for myelin synthesis. Ketogenic diet rescues myelin in ACLY-deficient mice. Connects to: N-Dimensional Gene Hypothesis, PIST polymorphic shifter, topological state machine, FAMM delay lines, waveprobe manifolds
29 lines
1 KiB
Text
29 lines
1 KiB
Text
created: 20260506230000000
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modified: 20260506230000000
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tags: ResearchStack Compression Hutter CAD Geometry MengerSponge GabrielHorn
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title: Pathological Menger-Horn Composite
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type: text/vnd.tiddlywiki
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! Pathological Menger-Horn Composite
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Composite manifold: Menger sponge nodes mounted as structural attachment points
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along Gabriel's horn backbone (x-axis curve y = 1/x). PIST shell coordinates are
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nested inside each sponge node.
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The horn provides infinite surface area with finite volume — the byte container
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surface. The Menger sponge provides fractal addressing (Hausdorff dimension
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~2.7268) for routing between shells. Together they form one of three pathological
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manifolds for Hutter Prize compression-geometry folding.
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Composite structure is Torus-Menger-Horn, as referenced in
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[[04_Hutter_Prize_Equation.md|Hutter Prize Equation §Torus-Menger-Horn folding]].
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!! Links
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* [[Hutter Prize Compression]]
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* [[Pathological Manifold Torus]]
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* [[Pathological PIST Shells]]
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!! Durable Source
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`../../3-Mathematical-Models/cad_models/composite_manifold.scad`
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