Research-Stack/6-Documentation/tiddlywiki-local/wiki/tiddlers/Pathological Menger-Horn Composite.tid
Brandon Schneider af97d84573 ingest: MS myelin glucose signaling article (2026-05-04)
Brain glucose levels regulate OPC fate: high glucose → proliferation,
low glucose → maturation. Acetyl-CoA from glucose drives histone
acetylation for OPC division; ketone bodies substitute for myelin
synthesis. Ketogenic diet rescues myelin in ACLY-deficient mice.

Connects to: N-Dimensional Gene Hypothesis, PIST polymorphic shifter,
topological state machine, FAMM delay lines, waveprobe manifolds
2026-05-06 23:53:20 -05:00

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created: 20260506230000000
modified: 20260506230000000
tags: ResearchStack Compression Hutter CAD Geometry MengerSponge GabrielHorn
title: Pathological Menger-Horn Composite
type: text/vnd.tiddlywiki
! Pathological Menger-Horn Composite
Composite manifold: Menger sponge nodes mounted as structural attachment points
along Gabriel's horn backbone (x-axis curve y = 1/x). PIST shell coordinates are
nested inside each sponge node.
The horn provides infinite surface area with finite volume — the byte container
surface. The Menger sponge provides fractal addressing (Hausdorff dimension
~2.7268) for routing between shells. Together they form one of three pathological
manifolds for Hutter Prize compression-geometry folding.
Composite structure is Torus-Menger-Horn, as referenced in
[[04_Hutter_Prize_Equation.md|Hutter Prize Equation §Torus-Menger-Horn folding]].
!! Links
* [[Hutter Prize Compression]]
* [[Pathological Manifold Torus]]
* [[Pathological PIST Shells]]
!! Durable Source
`../../3-Mathematical-Models/cad_models/composite_manifold.scad`